Iron chelation alleviates multiple pathophysiological pathways in a rat model of cardiac pressure overload.

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作者： Mihai,Lupu ^[1] ; Camelia Alexandra,Coada ^[2] ; Diana-Valentina,Tudor ^[1] ; Ioana,Baldea ^[1] ; Adrian,Florea ^[3] ; Vlad-Alexandru,Toma ^[4] ; Ana,Lupsor ^[1] ; Remus,Moldovan ^[1] ; Nicoleta,Decea ^[1] ; Gabriela Adriana,Filip ^[1]

作者单位： Iuliu Hatieganu University of Medicine and Pharmacy, Dept. of Physiology, Cluj-Napoca, Romania. ^[1] Iuliu Hatieganu University of Medicine and Pharmacy, Dept. of Molecular Sciences, 400394, Cluj-Napoca, Romania; University of Bologna, Department of Medical and Surgical Sciences (DIMEC), 40138, Bologna, Italy. ^[2] Iuliu Hatieganu University of Medicine and Pharmacy, Dept. of Cell and Molecular Biology, Cluj-Napoca, Romania. Electronic address: aflorea@umfcluj.ro. ^[3] Babeș-Bolyai University, Department of Molecular Biology and Biotechnologies, Clinicilor Street No. 4-6, 400000, Cluj-Napoca, Cluj County, Romania; Institute of Biological Research, Republicii Street No. 48, 400015, Cluj-Napoca, Cluj County, Romania. ^[4]

关键词 Chelation Chronic heart failure Hypertension Iron metabolism Lipid peroxidation

主题词大鼠(Rats);动物(Animals);铁超负荷(Iron Overload);铁螯合剂(Iron Chelating Agents);铁(Iron);心力衰竭, 收缩性(Heart Failure, Systolic);炎症(Inflammation)

DOI 10.1016/j.freeradbiomed.2023.02.018

PMID 36822542

发布时间 2023-05-11

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