Modeling Alzheimer's disease in primary neurons reveals DNA damage response coupled with MAPK-DLK signaling in wild-type tau-induced neurodegeneration.

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第一作者： Sanming,Li

第一单位： Department of Anesthesiology, Critical Care and Pain Medicine, McGovern Medical School, University of Texas Health Science Center at Houston, Houston, TX 77030, USA.

作者： Sanming,Li ^[1] ; Ethan R,Roy ^[1] ; Yanyu,Wang ^[1] ; Trent,Watkins ^[2] ; Wei,Cao ^[1]

作者单位： Department of Anesthesiology, Critical Care and Pain Medicine, McGovern Medical School, University of Texas Health Science Center at Houston, Houston, TX 77030, USA. ^[1] Department of Neurosurgery, Baylor College of Medicine, Houston, TX, USA. Current address: Department of Neurology, University of California at San Francisco, San Francisco, CA 94158 USA. ^[2]

关键词 Alzheimer Axonal degeneration DLK DNA damage MAP kinase Neurodegeneration Tauopathy

DOI 10.21203/rs.3.rs-2617457/v1

PMID 36945524

发布时间 2024-05-09

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Modeling Alzheimer's disease in primary neurons reveals DNA damage response coupled with MAPK-DLK signaling in wild-type tau-induced neurodegeneration.

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Modeling Alzheimer's disease in primary neurons reveals DNA damage response coupled with MAPK-DLK signaling in wild-type tau-induced neurodegeneration.

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