The β3-subunit modulates the effect of venom peptides ProTx-II and OD1 on Na<sub>V</sub> 1.7 gating.

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作者： Samantha C,Salvage ^[1] ; Taufiq,Rahman ^[2] ; David A,Eagles ^[3] ; Johanna S,Rees ^[4] ; Glenn F,King ^[1] ; Christopher L-H,Huang ^[3] ; Antony P,Jackson ^[4]

作者单位： Department of Biochemistry, University of Cambridge, Cambridge, UK. ^[1] Department of Pharmacology, University of Cambridge, Cambridge, UK. ^[2] Institute of Molecular Bioscience, University of Queensland, Brisbane, QLD, Australia. ^[3] Australian Research Council Centre of Excellence for Innovations in Peptide and Protein Science, The University of Queensland, Brisbane, QLD, Australia. ^[4] Department of Physiology, Development and Neuroscience, University of Cambridge, Cambridge, UK. ^[5]

关键词 NaV1.7 OD1 ProTx-II pain voltage-gated sodium channel β3-subunit

主题词人类(Humans);毒液类(Venoms);肽类(Peptides);镇痛药(Analgesics);疼痛(Pain)

DOI 10.1002/jcp.31018

PMID 37042220

发布时间 2024-03-22

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