Toll-like Receptor-9 (TLR-9) Signaling Is Crucial for Inducing Protective Immunity following Immunization with Genetically Modified Live Attenuated <i>Leishmania</i> Parasites.

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作者： Parna,Bhattacharya ^[1] ; Sreenivas,Gannavaram ^[1] ; Nevien,Ismail ^[1] ; Ankit,Saxena ^[2] ; Pradeep K,Dagur ^[3] ; Adovi,Akue ^[4] ; Mark,KuKuruga ^[4] ; Hira L,Nakhasi ^[1]

作者单位： Division of Emerging and Transfusion Transmitted Disease, Center for Biologics Evaluation and Research Food and Drug Administration, Silver Spring, MD 20993, USA. ^[1] Immune Monitoring Shared Resource, Rutgers, Cancer Institute of New Jersey, New Brunswick, NJ 08901, USA. ^[2] Flow Cytometry Core, National Heart, Lung, and Blood Institute, National Institutes of Health, Bethesda, MD 20892, USA. ^[3] Division of Bacterial, Parasitic, and Allergenic Products, Center for Biologics Evaluation and Research Food and Drug Administration, Silver Spring, MD 20993, USA. ^[4]

关键词 costimulatory molecules cytokines dendritic cells innate immunity myeloid differentiation primary response 88 nuclear factor-kappa B visceral leishmaniasis

DOI 10.3390/pathogens12040534

PMID 37111420

发布时间 2023-04-30

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Pathogens (Basel, Switzerland)

2023年12卷4期

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Toll-like Receptor-9 (TLR-9) Signaling Is Crucial for Inducing Protective Immunity following Immunization with Genetically Modified Live Attenuated <i>Leishmania</i> Parasites.

Pathogens (Basel, Switzerland)

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Pathogens (Basel, Switzerland)

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Toll-like Receptor-9 (TLR-9) Signaling Is Crucial for Inducing Protective Immunity following Immunization with Genetically Modified Live Attenuated <i>Leishmania</i> Parasites.

Pathogens (Basel, Switzerland)

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Pathogens (Basel, Switzerland)

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