HAPSTR1 localizes HUWE1 to the nucleus to limit stress signaling pathways.

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第一作者： Julie K,Monda

第一单位： School of Biological Sciences, Department of Cell and Developmental Biology, University of California, San Diego, La Jolla, CA 92093, USA.

作者： Julie K,Monda ^[1] ; Xuezhen,Ge ^[1] ; Moritz,Hunkeler ^[2] ; Katherine A,Donovan ^[2] ; Michelle W,Ma ^[2] ; Cyrus Y,Jin ^[3] ; Marilyn,Leonard ^[1] ; Eric S,Fischer ^[2] ; Eric J,Bennett ^[4]

作者单位： School of Biological Sciences, Department of Cell and Developmental Biology, University of California, San Diego, La Jolla, CA 92093, USA. ^[1] Department of Cancer Biology, Dana-Farber Cancer Institute, Boston, MA 02215, USA; Department of Biological Chemistry and Molecular Pharmacology, Harvard Medical School, Boston, MA 02115, USA. ^[2] Department of Cancer Biology, Dana-Farber Cancer Institute, Boston, MA 02215, USA. ^[3] School of Biological Sciences, Department of Cell and Developmental Biology, University of California, San Diego, La Jolla, CA 92093, USA. Electronic address: e1bennett@ucsd.edu. ^[4]

关键词 CP: Cell biology CP: Molecular biology HAPSTR1/TAPR1/C16orf72 HUWE1 protein quality control stress signaling ubiquitin

主题词泛素蛋白连接酶类(Ubiquitin-Protein Ligases);泛素(Ubiquitin);细胞系(Cell Line);DNA修复(DNA Repair);细胞核(Cell Nucleus);信号传导(Signal Transduction)

DOI 10.1016/j.celrep.2023.112496

PMID 37167062

发布时间 2023-06-20

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准确一般字义偏差语意偏离译文作用小提交