HBSP improves kidney ischemia-reperfusion injury and promotes repair in properdin deficient mice <i>via</i> enhancing phagocytosis of tubular epithelial cells.

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第一作者： Yuanyuan,Wu

第一单位： Department of Pathology, Medical School of Nantong University, Nantong, China.

作者： Yuanyuan,Wu ^[1] ; Lili,Huang ^[2] ; Weili,Sai ^[3] ; Fei,Chen ^[4] ; Yu,Liu ^[3] ; Cheng,Han ^[3] ; Joanna M,Barker ^[3] ; Zinah D,Zwaini ^[5] ; Mark P,Lowe ^[6] ; Nigel J,Brunskill ^[5] ; Bin,Yang ^[2]

作者单位： Department of Pathology, Medical School of Nantong University, Nantong, China. ^[1] Department of Cardiovascular Sciences, College of Life Sciences, University of Leicester, University Hospitals of Leicester NHS Trust, Leicester, United Kingdom. ^[2] Nantong-Leicester Joint Institute of Kidney Science, Nephrology, Affiliated Hospital of Nantong University, Nantong, China. ^[3] Research Center of Clinical Medicine, Affiliated Hospital of Nantong University, Nantong, China. ^[4] School of Chemistry, University of Leicester, Leicester, United Kingdom. ^[5] Department of Respiratory Sciences, College of Life Sciences, University of Leicester, Leicester, United Kingdom. ^[6]

关键词 HBSP innate repair receptor ischaemia-reperfusion injury phagocytosis properdin repair tubular epithelial cells