Loss of the mitochondrial protein Abcb10 results in altered arginine metabolism in MEL and K562 cells and nutrient stress signaling through ATF4.

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作者： Marisa,Miljkovic ^[1] ; Alexandra,Seguin ^[1] ; Xuan,Jia ^[1] ; James E,Cox ^[2] ; Jonathan Leon,Catrow ^[3] ; Hector,Bergonia ^[4] ; John D,Phillips ^[5] ; W Zac,Stephens ^[1] ; Diane M,Ward ^[6]

作者单位： Division of Microbiology and Immunology, Department of Pathology, University of Utah School of Medicine, Salt Lake City, Utah, USA. ^[1] Department of Biochemistry, University of Utah School of Medicine, Salt Lake City, Utah, USA; Metabolomics Core Research Facility, University of Utah School of Medicine, Salt Lake City, Utah, USA. ^[2] Metabolomics Core Research Facility, University of Utah School of Medicine, Salt Lake City, Utah, USA. ^[3] Iron and Heme Core Research Facility, University of Utah School of Medicine, Salt Lake City, Utah, USA. ^[4] Division of Hematology, Department of Medicine, University of Utah School of Medicine, Salt Lake City, Utah, USA. ^[5] Division of Microbiology and Immunology, Department of Pathology, University of Utah School of Medicine, Salt Lake City, Utah, USA. Electronic address: diane.mcveyward@path.utah.edu. ^[6]

关键词 arginine differentiation erythroid metabolism nutrient transporter