A clinicopathological analysis of supratentorial ependymoma, ZFTA fusion-positive: utility of immunohistochemical detection of CDKN2A alterations and characteristics of the immune microenvironment.

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作者： Naohito,Hashimoto ^[1] ; Tomonari,Suzuki ^[2] ; Keisuke,Ishizawa ^[1] ; Sumihito,Nobusawa ^[3] ; Hideaki,Yokoo ^[3] ; Ryo,Nishikawa ^[2] ; Masanori,Yasuda ^[4] ; Atsushi,Sasaki ^[5]

作者单位： Department of Pathology, Saitama Medical University Hospital, 38 Morohongou, Moroyama, Saitama, 350-0495, Japan. ^[1] Department of Neuro-Oncology/Neurosurgery, Saitama Medical University International Medical Center, Hidaka, Saitama, Japan. ^[2] Department of Human Pathology, Gunma University Graduate School of Medicine, Maebashi, Gunma, Japan. ^[3] Department of Pathology, Saitama Medical University International Medical Center, Hidaka, Saitama, Japan. ^[4] Department of Pathology, Saitama Medical University Hospital, 38 Morohongou, Moroyama, Saitama, 350-0495, Japan. asasaki@saitama-med.ac.jp. ^[5]

关键词 CDKN2A homozygous deletion MTAP-p16-supratentorial ependymoma Tumor-associated macrophages ZFTA fusion-positive

主题词人类(Humans);周期素依赖激酶抑制剂p16(Cyclin-Dependent Kinase Inhibitor p16);室管膜瘤(Ependymoma);脑肿瘤(Brain Neoplasms);肿瘤微环境(Tumor Microenvironment)

DOI 10.1007/s10014-023-00464-7

PMID 37322295

发布时间 2023-07-04

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