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Moderate intrinsic phenotypic alterations in <i>C9orf72</i> ALS/FTD iPSC-microglia despite the presence of C9orf72 pathological features.

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作者单位: Department of Translational Neuroscience, Barrow Neurological Institute, Phoenix, AZ, United States. [1] Neurogenomics Division, Translational Genomics Research Institute, Phoenix, AZ, United States. [2] Center for Neural Science and Medicine, Cedars-Sinai Medical Center, Regenerative Medicine Institute, Los Angeles, CA, United States. [3] Department of Translational Neuroscience, Barrow Neurological Institute, Phoenix, AZ, United States.;School of Life Sciences, Arizona State University, Tempe, AZ, United States. [4] Department of Neurobiology and Behavior, University of California, Irvine, Irvine, CA, United States.;Sue and Bill Gross Stem Cell Research Center, University of California, Irvine, Irvine, CA, United States.;Institute for Memory Impairments and Neurological Disorders, University of California, Irvine, Irvine, CA, United States. [5] UK Dementia Research Institute, The University of Edinburgh, Edinburgh, United Kingdom.;Centre for Brain Discovery Sciences, The University of Edinburgh, Edinburgh, United Kingdom. [6] Department of Neurology, University of Massachusetts Medical School, Worcester, MA, United States. [7] Department of Neurobiology, University of Pittsburgh School of Medicine, Pittsburgh, PA, United States. [8] Department of Stem Cell Biology Regenerative Medicine, USC Keck School of Medicine, Los Angeles, CA, United States. [9] Department of Neuroscience, Mayo Clinic, Jacksonville, FL, United States.;Mayo Clinic Graduate School of Biomedical Sciences, Mayo Clinic, Jacksonville, FL, United States. [10] Center for Neural Science and Medicine, Cedars-Sinai Medical Center, Regenerative Medicine Institute, Los Angeles, CA, United States.;Department of Neurology, Cedars-Sinai Medical Center, Los Angeles, CA, United States. [11]
DOI 10.3389/fncel.2023.1179796
PMID 37346371
发布时间 2024-12-16
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