Itaconate attenuates autoimmune hepatitis via PI3K/AKT/mTOR pathway-mediated inhibition of dendritic cell maturation and autophagy.

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第一作者： Qiyu,Zhang

第一单位： The First School of Clinical Medicine, Lanzhou University, 730000 Lanzhou, China.

作者： Qiyu,Zhang ^[1] ; Yang,Luo ^[2] ; Qiuxia,Zheng ^[3] ; Haixia,Zhao ^[4] ; Xiaofeng,Wei ^[5] ; Xun,Li ^[1]

作者单位： The First School of Clinical Medicine, Lanzhou University, 730000 Lanzhou, China. ^[1] Department of General Surgery, The First Hospital of Lanzhou University, 730000 Lanzhou, China. ^[2] Hepatopancreatobiliary Surgery Institute of Gansu Province, The First Hospital of Lanzhou University, 730000 Lanzhou, China. ^[3] Key Laboratory of Biotherapy and Regenerative Medicine of Gansu Province, The First Hospital of Lanzhou University, 730000 Lanzhou, China. ^[4] Medical Frontier Innovation Research Center, The First Hospital of Lanzhou University, 730000 Lanzhou, China. ^[5]

关键词 Autoimmune hepatitis Autophagy Dendritic cell Itaconate PI3K/AKT/mTOR

DOI 10.1016/j.heliyon.2023.e17551

PMID 37449121

发布时间 2023-07-18

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2023年9卷7期

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Itaconate attenuates autoimmune hepatitis via PI3K/AKT/mTOR pathway-mediated inhibition of dendritic cell maturation and autophagy.

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Itaconate attenuates autoimmune hepatitis via PI3K/AKT/mTOR pathway-mediated inhibition of dendritic cell maturation and autophagy.

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