Transcriptional profiling of rare acantholytic disorders suggests common mechanisms of pathogenesis.

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作者： Quinn R,Roth-Carter ^[1] ; Hope E,Burks ^[1] ; Ziyou,Ren ^[2] ; Jennifer L,Koetsier ^[1] ; Lam C,Tsoi ^[3] ; Paul W,Harms ^[4] ; Xianying,Xing ^[5] ; Joseph,Kirma ^[3] ; Robert M,Harmon ^[6] ; Lisa M,Godsel ^[3] ; Abbey L,Perl ^[3] ; Johann E,Gudjonsson ^[1] ; Kathleen J,Green ^[1]

作者单位： Department of Pathology, and. ^[1] Department of Dermatology, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, USA. ^[2] Department of Dermatology. ^[3] Department of Computational Medicine & Bioinformatics. ^[4] Department of Biostatistics, and. ^[5] Department of Pathology, University of Michigan, Ann Arbor, Michigan, USA. ^[6] Robert H. Lurie Comprehensive Cancer Center, Northwestern University, Chicago, Illinois, USA. ^[7]

关键词 Bioinformatics Cytoskeleton Dermatology Genetic diseases

主题词人类(Humans);肌动蛋白类(Actins);皮肤(Skin);皮肤棘层松解(Acantholysis);皮肤疾病(Skin Diseases)

DOI 10.1172/jci.insight.168955

PMID 37471166

发布时间 2024-02-10

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JCI insight

2023年8卷16期

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Transcriptional profiling of rare acantholytic disorders suggests common mechanisms of pathogenesis.

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Transcriptional profiling of rare acantholytic disorders suggests common mechanisms of pathogenesis.

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