Disulfide-HMGB1 signals through TLR4 and TLR9 to induce inflammatory macrophages capable of innate-adaptive crosstalk in human liver transplantation.

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第一作者： Allyson Q,Terry

第一单位： Department of Pathology and Laboratory Medicine, David Geffen School of Medicine at UCLA, Los Angeles, California, USA.

作者： Allyson Q,Terry ^[1] ; Hidenobu,Kojima ^[2] ; Rebecca A,Sosa ^[1] ; Fady M,Kaldas ^[2] ; Jackson L,Chin ^[3] ; Ying,Zheng ^[1] ; Bita V,Naini ^[1] ; Daisuke,Noguchi ^[2] ; Jessica,Nevarez-Mejia ^[1] ; Yi-Ping,Jin ^[1] ; Ronald W,Busuttil ^[2] ; Aaron S,Meyer ^[3] ; David W,Gjertson ^[4] ; Jerzy W,Kupiec-Weglinski ^[5] ; Elaine F,Reed ^[6]

作者单位： Department of Pathology and Laboratory Medicine, David Geffen School of Medicine at UCLA, Los Angeles, California, USA. ^[1] Division of Liver and Pancreas Transplantation, Department of Surgery, David Geffen School of Medicine at UCLA, Los Angeles, California, USA. ^[2] Department of Bioengineering, Samueli School of Engineering at UCLA, Los Angeles, California, USA. ^[3] Department of Pathology and Laboratory Medicine, David Geffen School of Medicine at UCLA, Los Angeles, California, USA; Department of Biostatistics, Fielding School of Public Health at UCLA, Los Angeles, California, USA. ^[4] Department of Pathology and Laboratory Medicine, David Geffen School of Medicine at UCLA, Los Angeles, California, USA; Division of Liver and Pancreas Transplantation, Department of Surgery, David Geffen School of Medicine at UCLA, Los Angeles, California, USA. ^[5] Department of Pathology and Laboratory Medicine, David Geffen School of Medicine at UCLA, Los Angeles, California, USA. Electronic address: ereed@mednet.ucla.edu. ^[6]

关键词 TLR activation alloimmunity disulfide-HMGB1 ischemia-reperfusion injury pro-inflammatory macrophage