Advanced glycation end products impair coronary artery BK channels via AMPK/Akt/FBXO32 signaling pathway.

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作者： Xiao-Yan,Li ^[1] ; Ling-Ling,Qian ^[1] ; Ying,Wu ^[1] ; Yu-Min,Zhang ^[1] ; Shi-Peng,Dang ^[1] ; Xiao-Yu,Liu ^[1] ; Xu,Tang ^[1] ; Cun-Yu,Lu ^[1] ; Ru-Xing,Wang ^[1]

作者单位： Department of Cardiology, The Affiliated Wuxi People's Hospital of Nanjing Medical University, Wuxi, China. ^[1]

关键词 Advanced glycation end products F-box protein 32 adenosine monophosphate activated protein kinase diabetes mellitus large conductance calcium activated potassium channel protein kinase B

主题词大鼠(Rats);人类(Humans);动物(Animals);冠状血管(Coronary Vessels);大电导钙激活钾通道(Large-Conductance Calcium-Activated Potassium Channels);AMP活化蛋白激酶类(AMP-Activated Protein Kinases);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);糖尿病, 实验性(Diabetes Mellitus, Experimental);信号传导(Signal Transduction);肌细胞, 平滑肌(Myocytes, Smooth Muscle);肌蛋白质类(Muscle Proteins);SKP Cullin F-框蛋白质连接酶类(SKP Cullin F-Box Protein Ligases)

DOI 10.1177/14791641231197107

PMID 37592725

发布时间 2023-11-08

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Diabetes & vascular disease research

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Advanced glycation end products impair coronary artery BK channels via AMPK/Akt/FBXO32 signaling pathway.

Diabetes & vascular disease research

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Diabetes & vascular disease research

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Advanced glycation end products impair coronary artery BK channels via AMPK/Akt/FBXO32 signaling pathway.

Diabetes & vascular disease research

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Diabetes & vascular disease research

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