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Gamma-patterned sensory stimulation reverses synaptic plasticity deficits in rat models of early Alzheimer's disease.

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第一作者： Yin,Yang

第一单位： Department of Pharmacology and Therapeutics, Trinity College Institute of Neuroscience, Trinity College, Dublin 2, Ireland.;Department of Physiology and Neurobiology, School of Basic Medical Sciences, Zhengzhou University, Zhengzhou, China.

作者： Yin,Yang ^[1] ; Tomas,Ondrejcak ^[2] ; Neng-Wei,Hu ^[1] ; Sadia,Islam ^[3] ; Eugene,O'Rourke ^[4] ; Richard B,Reilly ^[5] ; Colm,Cunningham ^[3] ; Michael J,Rowan ^[2] ; Igor,Klyubin ^[2]

作者单位： Department of Pharmacology and Therapeutics, Trinity College Institute of Neuroscience, Trinity College, Dublin 2, Ireland.;Department of Physiology and Neurobiology, School of Basic Medical Sciences, Zhengzhou University, Zhengzhou, China. ^[1] Department of Pharmacology and Therapeutics, Trinity College Institute of Neuroscience, Trinity College, Dublin 2, Ireland. ^[2] School of Biochemistry and Immunology, Trinity Biomedical Sciences Institute, Trinity College Institute of Neuroscience, Trinity College, Dublin 2, Ireland. ^[3] Department of Electronic and Electrical Engineering, Trinity College Institute of Neuroscience, Trinity College, Dublin 2, Ireland. ^[4] School of Medicine, Trinity College Institute of Neuroscience, Trinity College, Dublin 2, Ireland. ^[5]

关键词 40?Hz sensory stimulation Alzheimer's disease aqueous brain extract amyloid precursor protein transgenic rats beta-amyloid long-term potentiation tau

医学主题词动物(Animals);大鼠(Rats);阿尔茨海默病(Alzheimer Disease);神经元可塑性(Neuronal Plasticity);长时程增强(Long-Term Potentiation);大鼠, 转基因(Rats, Transgenic);淀粉样β蛋白前体(Amyloid beta-Protein Precursor)

DOI 10.1111/ejn.16129

PMID 37655756

发布时间 2023-09-20

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