A fatty acid-rich fraction of an endolichenic fungus <i>Phoma</i> sp. suppresses immune checkpoint markers via AhR/ARNT and ESR1.

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作者： Mücahit,Varlı ^[1] ; Men Thi,Ngo ^[2] ; Seong-Min,Kim ^[1] ; İsa,Taş ^[1] ; Rui,Zhou ^[1] ; Chathurika D B,Gamage ^[1] ; Sultan,Pulat ^[1] ; So-Yeon,Park ^[1] ; Nüzhet Cenk,Sesal ^[3] ; Jae-Seoun,Hur ^[4] ; Kyo Bin,Kang ^[2] ; Hangun,Kim ^[1]

作者单位： College of Pharmacy, Sunchon National University, 255 Jungang-ro, Sunchon, Jeonnam 57922, Republic of Korea. ^[1] College of Pharmacy, Sookmyung Women's University, 100 Cheongpa-ro 47 gil, Seoul 04310, Republic of Korea. ^[2] Faculty of Arts and Sciences, Department of Biology, Marmara University, Istanbul, Turkey. ^[3] Korean Lichen Research Institute, Sunchon National University, 255 Jungang-ro, Sunchon, Jeonnam 57922, Republic of Korea. ^[4]

关键词 Aryl hydrocarbon receptor Endolichenic fungi Estrogen receptor Immune checkpoint markers Natural product Programmed death ligand 1

DOI 10.1016/j.heliyon.2023.e19185

PMID 37662726

发布时间 2023-09-05

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Heliyon

2023年9卷9期

e19185页

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A fatty acid-rich fraction of an endolichenic fungus <i>Phoma</i> sp. suppresses immune checkpoint markers via AhR/ARNT and ESR1.

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A fatty acid-rich fraction of an endolichenic fungus <i>Phoma</i> sp. suppresses immune checkpoint markers via AhR/ARNT and ESR1.

Heliyon

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