Age-Dependent Increase in Tau Phosphorylation at Serine 396 in Huntington's Disease Prefrontal Cortex.

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作者： Tiziana,Petrozziello ^[1] ; Sommer S,Huntress ^[1] ; Ayleen L,Castillo-Torres ^[1] ; James P,Quinn ^[2] ; Theresa R,Connors ^[2] ; Corinne A,Auger ^[2] ; Alexandra N,Mills ^[1] ; Spencer E,Kim ^[1] ; Sophia,Liu ^[2] ; Farah,Mahmood ^[2] ; Adel,Boudi ^[2] ; Muzhou,Wu ^[3] ; Ellen,Sapp ^[2] ; Pia,Kivisäkk ^[2] ; Shekar R,Sunderesh ^[2] ; Mahmoud A,Pouladi ^[4] ; Steven E,Arnold ^[2] ; Bradley T,Hyman ^[2] ; H Diana,Rosas ^[2] ; Marian,DiFiglia ^[2] ; Ricardo,Mouro Pinto ^[5] ; Kimberly,Kegel-Gleason ^[2] ; Ghazaleh,Sadri-Vakili ^[1]

作者单位： Sean M. Healey & AMG Center for ALS at Mass General, MassGeneral Brigham, Boston, MA, USA. ^[1] Department of Neurology, MassGeneral Brigham, Boston, MA, USA. ^[2] Center for Genomic Medicine, MassGeneral Brigham, Boston, MA, USA. ^[3] Department of Medical Genetics, Centre for Molecular Medicine and Therapeutics, British Columbia Children's Hospital Research Institute, University of British Columbia, Vancouver, Canada. ^[4] Department of Neurology, MassGeneral Brigham, Boston, MA, USA.;Center for Genomic Medicine, MassGeneral Brigham, Boston, MA, USA.;Department of Neurology, Harvard Medical School, Boston, MA, USA. ^[5]

关键词 ESC-derived neurons Huntington’s disease fractionations mouse models neuronal stem cells phosphorylated tau plasma postmortem brain total tau