Interleukin-36γ is causative for liver damage upon infection with Rift Valley fever virus in type I interferon receptor-deficient mice.

导出 LinkOut

翻译打印收藏纠错

作者： Martina,Anzaghe ^[1] ; Marc A,Niles ^[1] ; Eugenia,Korotkova ^[1] ; Monica,Dominguez ^[1] ; Stefanie,Kronhart ^[1] ; Samira,Ortega Iannazzo ^[1] ; Ingo,Bechmann ^[2] ; Malte,Bachmann ^[3] ; Heiko,Mühl ^[3] ; Georg,Kochs ^[4] ; Zoe,Waibler ^[1]

作者单位： Division of Immunology, Paul-Ehrlich-Institut, Langen, Germany. ^[1] Medical Faculty, Institute for Anatomy, University Leipzig, Leipzig, Germany. ^[2] Pharmazentrum Frankfurt/ZAFES, University Hospital Frankfurt, Goethe-University Frankfurt am Main, Frankfurt am Main, Germany. ^[3] Institute of Virology, Medical Center, Faculty of Medicine, University of Freiburg, Freiburg, Germany. ^[4]

关键词 anti-inflammatory dysregulation immune pathology interleukin-36γ liver injury rift valley fever virus type I interferon

主题词动物(Animals);小鼠(Mice);白细胞介素1(Interleukin-1);肝(Liver);受体, 干扰素αβ(Receptor, Interferon alpha-beta);立夫特山谷热病毒(Rift Valley fever virus);裂谷热(Rift Valley Fever)

DOI 10.3389/fimmu.2023.1194733

PMID 37720217

发布时间 2023-09-28

翻译满意度评价：

准确一般字义偏差语意偏离译文作用小提交