Cross-species communication via agr controls phage susceptibility in Staphylococcus aureus.

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作者： Jingxian,Yang ^[1] ; Janine Zara,Bowring ^[1] ; Janes,Krusche ^[2] ; Esther,Lehmann ^[1] ; Benjamin Svejdal,Bejder ^[3] ; Stephanie Fulaz,Silva ^[1] ; Martin Saxtorph,Bojer ^[1] ; Tom,Grunert ^[4] ; Andreas,Peschel ^[2] ; Hanne,Ingmer ^[5]

作者单位： Department of Veterinary and Animal Sciences, Faculty of Health and Medical Sciences, University of Copenhagen, 1870 Copenhagen, Denmark. ^[1] Department of Infection Biology, Interfaculty Institute for Microbiology and Infection Medicine Tübingen (IMIT), University of Tübingen, 72076 Tübingen, Germany; Cluster of Excellence "Controlling Microbes to Fight Infections (CMFI)," German Center for Infection Research (DZIF), Tübingen, Germany. ^[2] Department of Drug Design and Pharmacology, Faculty of Health and Medical Sciences, University of Copenhagen, 2100 Copenhagen, Denmark. ^[3] Functional Microbiology, Institute of Microbiology, Department of Pathobiology, University of Veterinary Medicine, 1210 Vienna, Austria. ^[4] Department of Veterinary and Animal Sciences, Faculty of Health and Medical Sciences, University of Copenhagen, 1870 Copenhagen, Denmark. Electronic address: hi@sund.ku.dk. ^[5]

关键词 CP: Microbiology Stab20 WTA agr glycosylation phage tarM tarS