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Intestinal epithelial pH-sensing receptor GPR65 maintains mucosal homeostasis via regulating antimicrobial defense and restrains gut inflammation in inflammatory bowel disease.

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第一作者： Gengfeng,Li

第一单位： Center for IBD Research, Shanghai Tenth People's Hospital, School of Medicine, Tongji University, Shanghai, China.

作者： Gengfeng,Li ^[1] ; Jian,Lin ^[2] ; Xiang,Gao ^[1] ; Huiling,Su ^[3] ; Ritian,Lin ^[1] ; Han,Gao ^[1] ; Zhongsheng,Feng ^[1] ; Huili,Wu ^[4] ; Baisui,Feng ^[5] ; Keqiang,Zuo ^[1] ; Yingchuan,Li ^[1] ; Wei,Wu ^[1] ; Leilei,Fang ^[1] ; Zhanju,Liu ^[1]

作者单位： Center for IBD Research, Shanghai Tenth People's Hospital, School of Medicine, Tongji University, Shanghai, China. ^[1] Center for IBD Research, Shanghai Tenth People's Hospital, School of Medicine, Tongji University, Shanghai, China.;Department of Gastroenterology, Affiliated Hospital of Putian University, Putian, China. ^[2] Department of Gastroenterology, Linfen Central Hospital of Shanxi Medical University, Linfen, China. ^[3] Department of Gastroenterology, Zhengzhou Central Hospital Affiliated to Zhengzhou University, Zhengzhou, China. ^[4] Department of Gastroenterology, The Second Affiliated Hospital of Zhengzhou University, Zhengzhou, China. ^[5]

关键词 GPR65 antimicrobial defense antimicrobial peptide inflammatory bowel disease intestinal epithelial cell