Targeting pancreatic cancer metabolic dependencies through glutamine antagonism.

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作者： Joel,Encarnación-Rosado ^[1] ; Albert S W,Sohn ^[1] ; Douglas E,Biancur ^[1] ; Elaine Y,Lin ^[1] ; Victoria,Osorio-Vasquez ^[1] ; Tori,Rodrick ^[2] ; Diana,González-Baerga ^[1] ; Ende,Zhao ^[3] ; Yumi,Yokoyama ^[4] ; Diane M,Simeone ^[3] ; Drew R,Jones ^[2] ; Seth J,Parker ^[5] ; Robert,Wild ^[4] ; Alec C,Kimmelman ^[6]

作者单位： Perlmutter Cancer Center, New York University Grossman School of Medicine, New York, NY, USA.;Department of Radiation Oncology, New York University Grossman School of Medicine, New York, NY, USA. ^[1] Division of Advanced Research Technologies, New York University School of Medicine, New York, NY, USA. ^[2] Perlmutter Cancer Center, New York University Grossman School of Medicine, New York, NY, USA. ^[3] Dracen Pharmaceuticals, Inc., San Diego, CA, USA. ^[4] Department of Biochemistry & Molecular Biology, University of British Columbia, Vancouver, British Columbia, Canada. ^[5] Perlmutter Cancer Center, New York University Grossman School of Medicine, New York, NY, USA. Alec.Kimmelman@nyulangone.org.;Department of Radiation Oncology, New York University Grossman School of Medicine, New York, NY, USA. Alec.Kimmelman@nyulangone.org. ^[6]

主题词人类(Humans);谷氨酰胺(Glutamine);细胞系, 肿瘤(Cell Line, Tumor);胰腺肿瘤(Pancreatic Neoplasms);抗肿瘤药(Antineoplastic Agents);癌, 胰腺管(Carcinoma, Pancreatic Ductal);酶抑制剂(Enzyme Inhibitors)

DOI 10.1038/s43018-023-00647-3

PMID 37814010

发布时间 2024-10-23

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Nature cancer

2024年5卷1期

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Targeting pancreatic cancer metabolic dependencies through glutamine antagonism.

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Targeting pancreatic cancer metabolic dependencies through glutamine antagonism.

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