Investigating the Pathogenic Interplay of Alpha-Synuclein, Tau, and Amyloid Beta in Lewy Body Dementia: Insights from Viral-Mediated Overexpression in Transgenic Mouse Models.

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作者： Melina J,Lim ^[1] ; Suelen L,Boschen ^[1] ; Aishe,Kurti ^[2] ; Monica,Castanedes Casey ^[1] ; Virginia R,Phillips ^[1] ; John D,Fryer ^[1] ; Dennis,Dickson ^[3] ; Karen R,Jansen-West ^[1] ; Leonard,Petrucelli ^[1] ; Marion,Delenclos ^[1] ; Pamela J,McLean ^[1]

作者单位： Department of Neuroscience, Mayo Clinic, 4500 San Pablo Road, Jacksonville, FL 32224, USA. ^[1] Department of Neurosurgery, Mayo Clinic, 4500 San Pablo Road, Jacksonville, FL 32224, USA. ^[2] Department of Neuroscience, Mayo Clinic, 13400 E. Shea Blvd, Scottsdale, AZ 85259, USA. ^[3]

关键词 AAV Alzheimer’s disease Lewy body Lewy body dementia Parkinson’s disease alpha-synuclein amyloid beta mouse model tail vein tau

DOI 10.3390/biomedicines11102863

PMID 37893236

发布时间 2024-02-10

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Biomedicines

2023年11卷10期

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Investigating the Pathogenic Interplay of Alpha-Synuclein, Tau, and Amyloid Beta in Lewy Body Dementia: Insights from Viral-Mediated Overexpression in Transgenic Mouse Models.

Biomedicines

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Investigating the Pathogenic Interplay of Alpha-Synuclein, Tau, and Amyloid Beta in Lewy Body Dementia: Insights from Viral-Mediated Overexpression in Transgenic Mouse Models.

Biomedicines

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Biomedicines

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