Opposing roles for ADAMTS2 and ADAMTS14 in myofibroblast differentiation and function.

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作者： Edward P,Carter ^[1] ; Kubra K,Yoneten ^[2] ; Nuria,Gavara ^[3] ; Eleanor J,Tyler ^[4] ; Valentine,Gauthier ^[4] ; Elizabeth R,Murray ^[2] ; Peter,Ten Dijke ^[5] ; Angus J,Cameron ^[2] ; Oliver,Pearce ^[4] ; Richard P,Grose ^[2]

作者单位： Centre for Tumour Biology, Barts Cancer Institute, Queen Mary University of London, London, UK.;Department of Life Sciences, University of Bath, Bath, UK.;Centre for Therapeutic Innovation, Faculty of Science, University of Bath, Bath, UK. ^[1] Centre for Tumour Biology, Barts Cancer Institute, Queen Mary University of London, London, UK. ^[2] Unitat de Biofísica i Bioenginyeria, Facultat de Medicina i Ciències de la Salut, Universitat de Barcelona, Barcelona, Spain. ^[3] Centre for Tumour Microenvironment, Barts Cancer Institute, Queen Mary University of London, London, UK. ^[4] Oncode Institute and Department of Cell and Chemical Biology, Leiden University Medical Center, Leiden, The Netherlands. ^[5]

关键词 3D in vitro models TGFβ cancer invasion cancer-associated fibroblasts cellular cross talk extracellular matrix myofibroblast pancreatic cancer protease