The mediating role of AKT/ERK/JNK signaling on the malignant phenotype of microcystin-LR in gastric adenocarcinoma cells.

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第一作者： Yinghao,Liao

第一单位： Key Laboratory of Environmental Medicine Engineering, Ministry of Education, School of Public Health, Southeast University, Nanjing, 210009, China.

作者： Yinghao,Liao ^[1] ; Yali,Deng ^[2] ; Xiaojin,Yu ^[1] ; Peng,Zhang ^[3] ; Ran,Liu ^[4]

作者单位： Key Laboratory of Environmental Medicine Engineering, Ministry of Education, School of Public Health, Southeast University, Nanjing, 210009, China. ^[1] Key Laboratory of Environmental Medicine Engineering, Ministry of Education, School of Public Health, Southeast University, Nanjing, 210009, China; Center for Disease Control and Prevention of Huizhou, No. 10, Fumin Road, Huizhou, 516003, Guangdong, China. ^[2] Huzhou Center for Disease Prevention and Control, Huzhou, 313000, China. Electronic address: hzjkzp@163.com. ^[3] Key Laboratory of Environmental Medicine Engineering, Ministry of Education, School of Public Health, Southeast University, Nanjing, 210009, China. Electronic address: ranliu@seu.edu.cn. ^[4]

关键词 Gastric adenocarcinoma cells MC-LR Mediation effect PI3K/AKT/ERK Toxicity pathway

主题词动物(Animals);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);胃肿瘤(Stomach Neoplasms);MAP激酶信号系统(MAP Kinase Signaling System);微囊藻毒素类(Microcystins);腺癌(Adenocarcinoma)

DOI 10.1016/j.fct.2023.114174

PMID 37949205

发布时间 2023-12-16

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