RAB31 in glioma-derived endothelial cells promotes glioma cell invasion via extracellular vesicle-mediated enrichment of MYO1C.

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作者： Jinghao,Suo ^[1] ; Yuxin,Wang ^[2] ; Lin,Wang ^[1] ; Bojun,Qiu ^[2] ; Zhixing,Wang ^[1] ; An,Yan ^[2] ; Boqin,Qiang ^[1] ; Wei,Han ^[2] ; Xiaozhong,Peng ^[1]

作者单位： Department of Molecular Biology and Biochemistry, Institute of Basic Medical Sciences, Medical Primate Research Center, Neuroscience Center, Chinese Academy of Medical Sciences, School of Basic Medicine, Peking Union Medical College, Beijing, China. ^[1] State Key Laboratory of Common Mechanism Research for Major Diseases, Beijing, China. ^[2] State Key Laboratory of Respiratory Health and Multimorbidity, Beijing, China. ^[3] National Human Diseases Animal Model Resource Center, Beijing Engineering Research Center for Experimental Animal Models of Human Critical Diseases，Institute of Laboratory Animal Science, Chinese Academy of Medical Sciences & Peking Union Medical College, Beijing, China. ^[4]

关键词 MYO1C RAB31 endothelial cells extracellular vesicles glioma invasion

主题词人类(Humans);内皮细胞(Endothelial Cells);神经胶质瘤(Glioma);生物转运(Biological Transport);肌球蛋白Ⅰ型(Myosin Type I);rab GTP结合蛋白质类(rab GTP-Binding Proteins)

DOI 10.1002/2211-5463.13736

PMID 37953466

发布时间 2024-02-07

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