首页> Cells> Synergism between IL-33 and MRGPRX2/FcεRI Is Primarily Due to the Complementation of Signaling Modules, and Only Modestly Supplemented by Prolonged Activation of Selected Kinases.

Synergism between IL-33 and MRGPRX2/FcεRI Is Primarily Due to the Complementation of Signaling Modules, and Only Modestly Supplemented by Prolonged Activation of Selected Kinases.

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第一作者： Kristin,Franke

第一单位： Fraunhofer Institute for Translational Medicine and Pharmacology ITMP, Immunology and Allergology IA, 12203 Berlin, Germany.;Institute of Allergology, Charité-Universitätsmedizin Berlin, Corporate Member of Freie Universität Berlin and Humboldt Universität zu Berlin, Hindenburgdamm 30, 12203 Berlin, Germany.

作者： Kristin,Franke ^[1] ; Zhuoran,Li ^[1] ; Gürkan,Bal ^[1] ; Torsten,Zuberbier ^[1] ; Magda,Babina ^[1]

作者单位： Fraunhofer Institute for Translational Medicine and Pharmacology ITMP, Immunology and Allergology IA, 12203 Berlin, Germany.;Institute of Allergology, Charité-Universitätsmedizin Berlin, Corporate Member of Freie Universität Berlin and Humboldt Universität zu Berlin, Hindenburgdamm 30, 12203 Berlin, Germany. ^[1]

关键词 ERK FcεRI IL-33 JNK MRGPRX2 NF-κB PI3K TNF-α mast cells p38

医学主题词人类(Humans);NF-κB(NF-kappa B);信号传导(Signal Transduction);磷酸肌醇3-激酶类(Phosphatidylinositol 3-Kinases);神经组织蛋白质类(Nerve Tissue Proteins);受体, 神经肽(Receptors, Neuropeptide);受体, G-蛋白偶联(Receptors, G-Protein-Coupled)

DOI 10.3390/cells12232700

PMID 38067128

发布时间 2024-01-31

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