MiR-22-3p facilitates bone marrow mesenchymal stem cell osteogenesis and fracture healing through the SOSTDC1-PI3K/AKT pathway.

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作者： Chunqiu,Wang ^[1] ; Xinguo,Wang ^[2] ; Hui,Cheng ^[2] ; Jiahu,Fang ^[1]

作者单位： Department of Orthopedics, The First Affiliated Hospital of Nanjing Medical University, Nanjing, China. ^[1] Department of Orthopedics, Zhenjiang 359 Hospital, Zhenjiang, China. ^[2]

关键词 PI3K/AKT fracture healing miR-22-3p sclerostin domain-containing 1

主题词动物(Animals);人类(Humans);大鼠(Rats);衔接蛋白质类, 信号转导(Adaptor Proteins, Signal Transducing);细胞分化(Cell Differentiation);细胞, 培养的(Cells, Cultured);股骨骨折(Femoral Fractures);骨折愈合(Fracture Healing);间质干细胞(Mesenchymal Stem Cells);微RNAs(MicroRNAs);骨生成(Osteogenesis);磷酸肌醇3-激酶类(Phosphatidylinositol 3-Kinases);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt)

DOI 10.1111/iep.12500

PMID 38152045

发布时间 2025-01-04

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International journal of experimental pathology

2024年105卷2期

52-63页

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MiR-22-3p facilitates bone marrow mesenchymal stem cell osteogenesis and fracture healing through the SOSTDC1-PI3K/AKT pathway.

International journal of experimental pathology

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International journal of experimental pathology

相关期刊

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MiR-22-3p facilitates bone marrow mesenchymal stem cell osteogenesis and fracture healing through the SOSTDC1-PI3K/AKT pathway.

International journal of experimental pathology

相似文献

International journal of experimental pathology

相关期刊

检索历史清除