GABA<sub>A</sub> receptors as plausible molecular targets and mediators for taurine and homotaurine actions.

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作者： Pratap,Meera ^[1] ; Mikko,Uusi-Oukari ^[2] ; Gerald S,Lipshutz ^[3] ; Martin,Wallner ^[4]

作者单位： Department of Neurobiology, University of California, Los Angeles, Los Angeles, CA, United States. ^[1] Integrative Physiology and Pharmacology, Institute of Biomedicine, University of Turku, Turku, Finland. ^[2] Department of Surgery, University of California, Los Angeles, Los Angeles, CA, United States. ^[3] Molecular and Medical Pharmacology, University of California, Los Angeles, Los Angeles, CA, United States. ^[4] Intellectual and Developmental Disabilities Research Center, University of California, Los Angeles, Los Angeles, CA, United States. ^[5] Semel Institute for Neuroscience, University of California, Los Angeles, Los Angeles, CA, United States. ^[6]

关键词 Alzheimer’s disease GABA-mimetics GABAA receptor homotaurine inflammation taurine tramiprosate

DOI 10.3389/fphar.2023.1271203

PMID 38155909

发布时间 2024-02-10

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Frontiers in pharmacology

2023年14卷

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GABA<sub>A</sub> receptors as plausible molecular targets and mediators for taurine and homotaurine actions.

Frontiers in pharmacology

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Frontiers in pharmacology

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GABA<sub>A</sub> receptors as plausible molecular targets and mediators for taurine and homotaurine actions.

Frontiers in pharmacology

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Frontiers in pharmacology

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