Soluble CD83 modulates human-monocyte-derived macrophages toward alternative phenotype, function, and metabolism.

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作者： Katrin,Peckert-Maier ^[1] ; Andreas B,Wild ^[1] ; Laura,Sprißler ^[1] ; Maximilian,Fuchs ^[2] ; Philipp,Beck ^[1] ; Jean-Philippe,Auger ^[3] ; Pia,Sinner ^[1] ; Astrid,Strack ^[1] ; Petra,Mühl-Zürbes ^[1] ; Ntilek,Ramadan ^[1] ; Meik,Kunz ^[2] ; Gerhard,Krönke ^[4] ; Lena,Stich ^[3] ; Alexander,Steinkasserer ^[1] ; Dmytro,Royzman ^[1]

作者单位： Department of Immune Modulation, Universitätsklinikum Erlangen, Friedrich- Alexander Universität Erlangen-Nürnberg, Erlangen, Germany. ^[1] Fraunhofer Institute for Toxicology and Experimental Medicine (ITEM), Hannover, Germany. ^[2] Department of Internal Medicine 3 - Rheumatology and Immunology, Friedrich-Alexander University Erlangen-Nürnberg (FAU) and Universitätsklinikum Erlangen, Erlangen, Germany. ^[3] Chair of Medical Informatics, Friedrich-Alexander-Universität Erlangen-Nürnberg (FAU), Erlangen, Bavaria, Germany. ^[4]

关键词 LXR pathway alternative activation checkpoint molecule human-monocyte-derived macrophages soluble CD83

主题词人类(Humans);细胞分化(Cell Differentiation);巨噬细胞(Macrophages);表型(Phenotype);T淋巴细胞(T-Lymphocytes)

DOI 10.3389/fimmu.2023.1293828

PMID 38162675

发布时间 2024-03-29

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