Peripheral thickening of the sarcomeres and pointed end elongation of the thin filaments are both promoted by SALS and its formin interaction partners.

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第一作者： Dávid,Farkas

第一单位： Institute of Genetics, Biological Research Centre, Szeged, Hungary.

作者： Dávid,Farkas ^[1] ; Szilárd,Szikora ^[1] ; A S,Jijumon ^[1] ; Tamás F,Polgár ^[2] ; Roland,Patai ^[3] ; Mónika Ágnes,Tóth ^[4] ; Beáta,Bugyi ^[4] ; Tamás,Gajdos ^[5] ; Péter,Bíró ^[5] ; Tibor,Novák ^[5] ; Miklós,Erdélyi ^[5] ; József,Mihály ^[6]

作者单位： Institute of Genetics, Biological Research Centre, Szeged, Hungary. ^[1] Institute of Biophysics, Biological Research Centre, Szeged, Hungary.;Doctoral School of Theoretical Medicine, University of Szeged, Szeged, Hungary. ^[2] Institute of Biophysics, Biological Research Centre, Szeged, Hungary. ^[3] University of Pécs, Medical School, Department of Biophysics, Pécs, Hungary. ^[4] Department of Optics and Quantum Electronics, University of Szeged, Szeged, Hungary. ^[5] Institute of Genetics, Biological Research Centre, Szeged, Hungary.;University of Szeged, Department of Genetics, Szeged, Hungary. ^[6]

主题词动物(Animals);人类(Humans);肌节(Sarcomeres);肌动蛋白类(Actins);肌病, 杆状体(Myopathies, Nemaline);果蝇属(Drosophila)

DOI 10.1371/journal.pgen.1011117

PMID 38198522

发布时间 2024-10-23

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