Nucleocytoplasmic transport rates are regulated by cellular processes that modulate GTP availability.

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作者： Kelsey L,Scott ^[1] ; Charles T,Halfmann ^[1] ; Allison D,Hoefakker ^[2] ; Purboja,Purkayastha ^[3] ; Ting Ching,Wang ^[3] ; Tanmay P,Lele ^[4] ; Kyle J,Roux ^[5]

作者单位： Enabling Technologies Group, Sanford Research, Sioux Falls SD. ^[1] Enabling Technologies Group, Sanford Research, Sioux Falls SD.;Basic Biomedical Sciences, Sanford School of Medicine, University of South Dakota, Vermillion, SD. ^[2] Artie McFerrin Department of Chemical Engineering, Texas A&M University, College Station, Texas. ^[3] Artie McFerrin Department of Chemical Engineering, Texas A&M University, College Station, Texas.;Department of Biomedical Engineering, Texas A&M University, College Station, Texas.;Department of Translational Medical Sciences, Texas A&M University, Houston, Texas. ^[4] Enabling Technologies Group, Sanford Research, Sioux Falls SD.;Department of Pediatrics, Sanford School of Medicine, University of South Dakota, Sioux Falls, SD. ^[5]

关键词 GEVAL GTP LINuS Nucleocytoplasmic transport Ran GTPase glucocorticoid receptor nuclear pore complex

DOI 10.1101/2023.12.29.573651

PMID 38234722

发布时间 2024-07-29

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Nucleocytoplasmic transport rates are regulated by cellular processes that modulate GTP availability.

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Nucleocytoplasmic transport rates are regulated by cellular processes that modulate GTP availability.

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