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Development of primary osteoarthritis during aging in genetically diverse UM-HET3 mice.

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第一作者： Sher Bahadur,Poudel

第一单位： David B. Kriser Dental Center, Department of Molecular Pathobiology, New York University College of Dentistry, New York, NY.

作者： Sher Bahadur,Poudel ^[1] ; Ryan R,Ruff ^[2] ; Gozde,Yildirim ^[1] ; Richard A,Miller ^[3] ; David E,Harrison ^[4] ; Randy,Strong ^[5] ; Thorsten,Kirsch ^[6] ; Shoshana,Yakar ^[1]

作者单位： David B. Kriser Dental Center, Department of Molecular Pathobiology, New York University College of Dentistry, New York, NY. ^[1] David B. Kriser Dental Center, Biostatistics Core, Department of Epidemiology and Health Promotion, New York University College of Dentistry New York, NY 10010-4086. ^[2] Department of Pathology and Geriatrics Center, University of Michigan, Ann Arbor, MI, USA. ^[3] The Jackson Laboratory, Bar Harbor, ME, USA. ^[4] Geriatric Research, Education and Clinical Center and Research Service, South Texas Veterans Health Care System, San Antonio, TX, USA; Barshop Institute for Longevity and Aging Studies and Department of Pharmacology, The University of Texas Health Science Center, San Antonio, TX, USA. ^[5] Department of Orthopaedic Surgery, NYU Grossman School of Medicine, and Department of Biomedical Engineering, NYU Tandon School of Engineering, New York, NY. ^[6]

关键词 UM-HET3 antioxidants cartilage methylene blue mitoquinone osteoarthritis sub-chondral bone

DOI 10.21203/rs.3.rs-3858256/v1

PMID 38343826

发布时间 2024-06-17

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Development of primary osteoarthritis during aging in genetically diverse UM-HET3 mice.

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