Neutrophils are indispensable for adverse cardiac remodeling in heart failure.

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第一作者： Sergey,Antipenko

第一单位： Division of Cardiovascular Disease, University of Alabama at Birmingham, Birmingham, AL, USA.

作者： Sergey,Antipenko ^[1] ; Nicolas,Mayfield ^[1] ; Miki,Jinno ^[1] ; Matthias,Gunzer ^[2] ; Mohamed Ameen,Ismahil ^[3] ; Tariq,Hamid ^[3] ; Sumanth D,Prabhu ^[4] ; Gregg,Rokosh ^[5]

作者单位： Division of Cardiovascular Disease, University of Alabama at Birmingham, Birmingham, AL, USA. ^[1] Institute for Experimental Immunology and Imaging, University Hospital, University of Duisburg-Essen, Essen, Germany; Leibniz-Institute fur Analytische Wissenschaften - ISAS - e.V., Dortmund, Germany. ^[2] Division of Cardiovascular Disease, University of Alabama at Birmingham, Birmingham, AL, USA; Division of Cardiology, Washington University School of Medicine, St. Louis, MO, USA. ^[3] Division of Cardiovascular Disease, University of Alabama at Birmingham, Birmingham, AL, USA; Division of Cardiology, Washington University School of Medicine, St. Louis, MO, USA. Electronic address: prabhu@wustl.edu. ^[4] Division of Cardiovascular Disease, University of Alabama at Birmingham, Birmingham, AL, USA; Division of Cardiology, Washington University School of Medicine, St. Louis, MO, USA. Electronic address: rokosh@wustl.edu. ^[5]

关键词 Fibrosis Heart failure Inflammation LV remodeling NETosis Neutrophils