CD62E- and ROS-Responsive ETS Improves Cartilage Repair by Inhibiting Endothelial Cell Activation through OPA1-Mediated Mitochondrial Homeostasis.

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第一作者： Pengcheng,Tu

第一单位： Affiliated Hospital of Nanjing University of Chinese Medicine, Nanjing 210029, P.R. China.

作者： Pengcheng,Tu ^[1] ; Yalan,Pan ^[2] ; Lining,Wang ^[2] ; Bin,Li ^[2] ; Xiaoxian,Sun ^[3] ; Zhongqing,Liang ^[2] ; Mengmin,Liu ^[3] ; Zitong,Zhao ^[1] ; Chengjie,Wu ^[2] ; Jianwei,Wang ^[4] ; Zhifang,Wang ^[5] ; Yu,Song ^[2] ; Yafeng,Zhang ^[3] ; Yong,Ma ^[2] ; Yang,Guo ^[3]

作者单位： Affiliated Hospital of Nanjing University of Chinese Medicine, Nanjing 210029, P.R. China. ^[1] Laboratory of New Techniques of Restoration and Reconstruction of Orthopedics and Traumatology, Nanjing University of Chinese Medicine, Nanjing 210023, P.R. China. ^[2] School of Chinese Medicine, School of Integrated Chinese and Western Medicine, Nanjing University of Chinese Medicine, Nanjing 210023, P.R. China. ^[3] Key Laboratory of Acupuncture and Medicine Research of Ministry of Education, Nanjing University of Chinese Medicine, Nanjing 210023, China. ^[4] School of Acupuncture and Tuina, School of Health and Rehabilitation, Nanjing University of Chinese Medicine, Nanjing 210046, Jiangsu, China. ^[5] Jiangsu CM Clinical Innovation Center of Degenerative Bone & Joint Disease, Wuxi TCM Hospital Affiliated to Nanjing University of Chinese Medicine, Wuxi 214072, P.R. China. ^[6] Zhangjiagang TCM Hospital Affiliated to Nanjing University of Chinese Medicine, Zhangjiagang 215600, P.R. China. ^[7] Zhangjiagang First People's Hospital Affiliated to Soochow University, Zhangjiagang 215638, P.R. China. ^[8]

DOI 10.34133/bmr.0006

PMID 38439927

发布时间 2024-03-06

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Biomaterials research

2024年28卷

0006页

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CD62E- and ROS-Responsive ETS Improves Cartilage Repair by Inhibiting Endothelial Cell Activation through OPA1-Mediated Mitochondrial Homeostasis.

Biomaterials research

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Biomaterials research

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CD62E- and ROS-Responsive ETS Improves Cartilage Repair by Inhibiting Endothelial Cell Activation through OPA1-Mediated Mitochondrial Homeostasis.

Biomaterials research

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Biomaterials research

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