首页> Research square> SM22α deficiency: promoting vascular fibrosis via SRF-SMAD3-mediated activation of <i>Col1a2</i> transcription following arterial injury.

SM22α deficiency: promoting vascular fibrosis via SRF-SMAD3-mediated activation of <i>Col1a2</i> transcription following arterial injury.

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第一作者： Jianbin,Shen

第一单位： Wayne State University School of Medicine.

作者： Jianbin,Shen ^[1] ; Donghong,Ju ^[2] ; Shichao,Wu ^[2] ; Jiawei,Zhao ^[2] ; Lucynda,Pham ^[2] ; Alejandro,Ponce ^[2] ; Maozhou,Yang ^[3] ; Hui Joyce,Li ^[4] ; Kezhong,Zhang ^[2] ; Zhe,Yang ^[2] ; Youming,Xie ^[2] ; Li,Li ^[2]

作者单位： Wayne State University School of Medicine. ^[1] Wayne State University. ^[2] Henry Ford Health System. ^[3] University of Massachusetts Chan Medical School. ^[4]

关键词 Cal1a2 SM22/Tagln SRF Smad3 transcription vascular fibrosis

DOI 10.21203/rs.3.rs-3941602/v1

PMID 38464061

发布时间 2026-05-11

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SM22α deficiency: promoting vascular fibrosis via SRF-SMAD3-mediated activation of <i>Col1a2</i> transcription following arterial injury.

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SM22α deficiency: promoting vascular fibrosis via SRF-SMAD3-mediated activation of <i>Col1a2</i> transcription following arterial injury.

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