换一批TMEM158 functions as an oncogene and promotes lung adenocarcinoma progression through the PI3K/AKT pathway via interaction with TWIST1.
第一作者：
Tao,Xu
第一单位：
Anhui Medical University, Hefei, 230000, China; Department of Cardiothoracic Surgery, The First People's Hospital of Wuhu, Wuhu, 241000, China. Electronic address: 272427802@qq.com.
作者：
医学主题词
人类(Humans);磷酸肌醇3-激酶类(Phosphatidylinositol 3-Kinases);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);癌基因(Oncogenes);肺肿瘤(Lung Neoplasms);细胞增殖(Cell Proliferation);细胞运动(Cell Movement);细胞系, 肿瘤(Cell Line, Tumor);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);核蛋白质类(Nuclear Proteins);膜蛋白质类(Membrane Proteins);肿瘤抑制蛋白质类(Tumor Suppressor Proteins)
DOI
10.1016/j.yexcr.2024.114010
PMID
38508329
发布时间
2024-04-08
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