Oxygen-Glucose Deprivation Increases NR4A1 Expression and Promotes Its Extranuclear Translocation in Mouse Astrocytes.

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第一作者： Kengo,Moriyama

第一单位： Department of Brain and Neuroscience, Tokyo Metropolitan Institute of Medical Science, Tokyo 156-8506, Japan.;Department of Pediatrics, Tokyo Medical and Dental University, Tokyo 113-8510, Japan.

作者： Kengo,Moriyama ^[1] ; Asako,Horino ^[2] ; Kuniko,Kohyama ^[2] ; Yasumasa,Nishito ^[3] ; Tomohiro,Morio ^[4] ; Hiroshi,Sakuma ^[2]

作者单位： Department of Brain and Neuroscience, Tokyo Metropolitan Institute of Medical Science, Tokyo 156-8506, Japan.;Department of Pediatrics, Tokyo Medical and Dental University, Tokyo 113-8510, Japan. ^[1] Department of Brain and Neuroscience, Tokyo Metropolitan Institute of Medical Science, Tokyo 156-8506, Japan. ^[2] Technology Research Division, Tokyo Metropolitan Institute of Medical Science, Tokyo 156-8506, Japan. ^[3] Department of Pediatrics, Tokyo Medical and Dental University, Tokyo 113-8510, Japan. ^[4]

关键词 CDIM8 HIF-1α NR4A1 NR4A3 mitochondria oxygen–glucose deprivation pexidartinib reoxygenation

DOI 10.3390/brainsci14030244

PMID 38539632

发布时间 2024-03-30

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Brain sciences

2024年14卷3期

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Oxygen-Glucose Deprivation Increases NR4A1 Expression and Promotes Its Extranuclear Translocation in Mouse Astrocytes.

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Oxygen-Glucose Deprivation Increases NR4A1 Expression and Promotes Its Extranuclear Translocation in Mouse Astrocytes.

Brain sciences

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Brain sciences

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