3-methyl-1H-indol-1-yl dimethylcarbamodithioate attenuates periodontitis through targeting MAPK signaling pathway-regulated mitochondrial function.

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第一作者： Yun,Jiang

第一单位： Institute of Stomatology, School and Hospital of Stomatology, Wenzhou Medical University, Wenzhou, China.

作者： Yun,Jiang ^[1] ; Xuekun,Ren ^[2] ; Jiajie,Mao ^[3] ; Jun,Zeng ^[4] ; Wanying,Jiang ^[1] ; Runqi,Zhou ^[5] ; Yue,Han ^[1] ; Hongning,Wang ^[2] ; Yixin,Mao ^[3] ; Xiaoyu,Sun ^[4] ; Zelin,Cao ^[1] ; Zengqiang,Song ^[1] ; Shengbin,Huang ^[1] ; Shufan,Zhao ^[2]

作者单位： Institute of Stomatology, School and Hospital of Stomatology, Wenzhou Medical University, Wenzhou, China. ^[1] Department of Prosthodontics, School and Hospital of Stomatology, Wenzhou Medical University, Wenzhou, China. ^[2] The Affiliated Hospital of Stomatology, School of Stomatology, Zhejiang University School of Medicine, Hangzhou, Zhejiang, China. ^[3] Key Laboratory of Oral Biomedical Research of Zhejiang Province, Hangzhou, Zhejiang, China. ^[4] Department of Endodontics, School and Hospital of Stomatology, Wenzhou Medical University, Wenzhou, China. ^[5] Department of Periodontics, School and Hospital of Stomatology, Wenzhou Medical University, Wenzhou, China. ^[6] Department of Biochemistry, School of Basic Medical Sciences, Wenzhou Medical University, Wenzhou, Zhejiang, China. ^[7] School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou, Zhejiang, China. ^[8] Department of Oral Maxillofacial Surgery, School and Hospital of Stomatology, Wenzhou Medical University, Wenzhou, China. ^[9]

关键词 inflammation macrophage mitochondria osteoclast differentiation periodontitis