Alpha-synuclein interaction with mitochondria is the final mechanism of ferroptotic death induced by erastin in SH-SY5Y cells.

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第一作者： Upasana,Ganguly

第一单位： Department of Biochemistry and Central Research Cell, Maharishi Markandeshwar Institute of Medical Sciences and Research, Maharishi Markandeshwar University (Deemed to be), Ambala, India.

作者： Upasana,Ganguly ^[1] ; Sukhpal,Singh ^[1] ; Aritri,Bir ^[2] ; Arindam,Ghosh ^[2] ; Sankha Shubhra,Chakrabarti ^[3] ; Reena V,Saini ^[4] ; Luciano,Saso ^[5] ; Marco,Bisaglia ^[6] ; Sasanka,Chakrabarti ^[1]

作者单位： Department of Biochemistry and Central Research Cell, Maharishi Markandeshwar Institute of Medical Sciences and Research, Maharishi Markandeshwar University (Deemed to be), Ambala, India. ^[1] Department of Biochemistry, Dr B. C. Roy Multi-Speciality Medical Research Centre, IIT Kharagpur, India. ^[2] Department of Geriatric Medicine, Institute of Medical Sciences, Banaras Hindu University, Varanasi, India. ^[3] Department of Biotechnology, Maharishi Markandeshwar Engineering College, Maharishi Markandeshwar University (Deemed to be), Ambala, India. ^[4] Department of Physiology and Pharmacology, Sapienza University of Rome, Rome, Italy. ^[5] Department of Biology, University of Padova, Padova, Italy. ^[6]

关键词 Mitochondria cyclosporine A ferrostatin-1 liproxstatin-1 mitochondrial permeability transition pore

主题词人类(Humans);α突触核蛋白(alpha-Synuclein);细胞死亡(Cell Death);细胞系, 肿瘤(Cell Line, Tumor);线粒体(Mitochondria);哌嗪类(Piperazines);活性氧(Reactive Oxygen Species)

DOI 10.1080/10715762.2024.2336563

PMID 38572725

发布时间 2024-05-03

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