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首页> European journal of pharmacology> Bradykinin attenuates endothelial-mesenchymal transition following cardiac ischemia-reperfusion injury.

Bradykinin attenuates endothelial-mesenchymal transition following cardiac ischemia-reperfusion injury.

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作者： Jinchao,Song ^[1] ; Jiankui,Du ^[2] ; Xing,Tan ^[3] ; Haiyan,Chen ^[4] ; Binhai,Cong ^[5]

作者单位： Department of Anesthesiology, Shidong Hospital Affiliated to the University of Shanghai for Science and Technology, Shanghai, China; Department of Physiology, Naval Medical University (Second Military Medical University), Shanghai, China. ^[1] Department of Physiology, Naval Medical University (Second Military Medical University), Shanghai, China. ^[2] Department of Physiology, Naval Medical University (Second Military Medical University), Shanghai, China; Department of Marine Biomedicine and Polar Medicine, Naval Medical Center, Naval Medical University (Second Military Medical University), Shanghai, China. ^[3] Department of Anesthesiology, Shidong Hospital Affiliated to the University of Shanghai for Science and Technology, Shanghai, China. ^[4] Department of Physiology, Naval Medical University (Second Military Medical University), Shanghai, China. Electronic address: congbinhai@smmu.edu.cn. ^[5]

关键词 Bradykinin Cardiac fibrosis Cardiac remodeling Endothelial-mesenchymal transition Nitric oxide

主题词人类(Humans);大鼠(Rats);动物(Animals);内皮细胞(Endothelial Cells);缓激肽(Bradykinin);心肌疾病(Cardiomyopathies);受体, 缓激肽(Receptors, Bradykinin);一氧化氮(Nitric Oxide);再灌注损伤(Reperfusion Injury);纤维化(Fibrosis);上皮-间质转化(Epithelial-Mesenchymal Transition)

DOI 10.1016/j.ejphar.2024.176556

PMID 38574840

发布时间 2024-04-22

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European journal of pharmacology

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