Pharmacological enhancement of cholinergic neurotransmission alleviates neuroinflammation and improves functional outcomes in a triple transgenic mouse model of Alzheimer's disease.

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作者： Antonio,Munafò ^[1] ; Anna Flavia,Cantone ^[1] ; Giulia,Di Benedetto ^[1] ; Sebastiano Alfio,Torrisi ^[2] ; Chiara,Burgaletto ^[1] ; Carlo Maria,Bellanca ^[1] ; Gabriella,Gaudio ^[1] ; Giuseppe,Broggi ^[2] ; Rosario,Caltabiano ^[1] ; Gian Marco,Leggio ^[3] ; Renato,Bernardini ^[3] ; Giuseppina,Cantarella ^[1]

作者单位： Department of Biomedical and Biotechnological Sciences, Section of Pharmacology, University of Catania, Catania, Italy. ^[1] Clinical Toxicology Unit, University Hospital of Catania, Catania, Italy. ^[2] Department of Medical, Surgical Sciences and Advanced Technologies "G.F. Ingrassia", Anatomic Pathology, University of Catania, Catania, Italy. ^[3]

关键词 amyloid plaques choline alphoscerate glial cells memory mouse brain neurodegeneration synapses

DOI 10.3389/fphar.2024.1386224

PMID 38595916

发布时间 2024-04-11

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Frontiers in pharmacology

2024年15卷

1386224页

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Pharmacological enhancement of cholinergic neurotransmission alleviates neuroinflammation and improves functional outcomes in a triple transgenic mouse model of Alzheimer's disease.

Frontiers in pharmacology

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Frontiers in pharmacology

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Pharmacological enhancement of cholinergic neurotransmission alleviates neuroinflammation and improves functional outcomes in a triple transgenic mouse model of Alzheimer's disease.

Frontiers in pharmacology

相似文献

Frontiers in pharmacology

相关期刊

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