ISGylation by HERCs facilitates STING activation.

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作者： Ying,Qin ^[1] ; Min,Wang ^[2] ; Xintong,Meng ^[1] ; Mengge,Wang ^[1] ; Haojia,Jiang ^[1] ; Yanjie,Gao ^[1] ; Jingxin,Li ^[1] ; Chunyuan,Zhao ^[1] ; Chaofeng,Han ^[3] ; Wei,Zhao ^[4] ; Xuexing,Zheng ^[5]

作者单位： Key Laboratory for Experimental Teratology of the Chinese Ministry of Education and Key Laboratory of Infection and Immunity of Shandong Province, School of Basic Medical Science, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, China. ^[1] Department of Virology, School of Public Health, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, China. ^[2] Department of Histology and Embryology and Shanghai Key Laboratory of Cell Engineering, Naval Medical University, Shanghai, China. ^[3] Key Laboratory for Experimental Teratology of the Chinese Ministry of Education and Key Laboratory of Infection and Immunity of Shandong Province, School of Basic Medical Science, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, China; Department of Histology and Embryology and Shanghai Key Laboratory of Cell Engineering, Naval Medical University, Shanghai, China. Electronic address: wzhao@sdu.edu.cn. ^[4] Department of Virology, School of Public Health, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, China. Electronic address: zhengxuexing@sdu.edu.cn. ^[5]

关键词 Antiviral innate immunity CP: Immunology HERCs ISGylation SARS-CoV-2 protein PLpro STING post-translational modifications