Improved multi-ancestry fine-mapping identifies <i>cis</i>-regulatory variants underlying molecular traits and disease risk.

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第一作者： Zeyun,Lu

第一单位： Center for Genetic Epidemiology, Keck School of Medicine, University of Southern California, Los Angeles, CA, USA.

作者： Zeyun,Lu ^[1] ; Xinran,Wang ^[1] ; Matthew,Carr ^[1] ; Artem,Kim ^[1] ; Steven,Gazal ^[2] ; Pejman,Mohammadi ^[3] ; Lang,Wu ^[4] ; Alexander,Gusev ^[5] ; James,Pirruccello ^[6] ; Linda,Kachuri ^[7] ; Nicholas,Mancuso ^[2]

作者单位： Center for Genetic Epidemiology, Keck School of Medicine, University of Southern California, Los Angeles, CA, USA. ^[1] Center for Genetic Epidemiology, Keck School of Medicine, University of Southern California, Los Angeles, CA, USA.;Department of Population and Public Health Sciences, Keck School of Medicine, University of Southern California, Los Angeles, CA, USA.;Department of Quantitative and Computational Biology, University of Southern California, Los Angeles, CA. ^[2] Center for Immunity and Immunotherapies, Seattle Children's Research Institute, Seattle, WA, USA.;Department of Pediatrics, University of Washington School of Medicine, Seattle, WA, USA.;Department of Genome Sciences, University of Washington, Seattle, WA, USA. ^[3] Cancer Epidemiology Division, Population Sciences in the Pacific Program, University of Hawai'i Cancer Center, University of Hawai'i at Mānoa, Honolulu, HI, USA. ^[4] Harvard Medical School and Dana-Farber Cancer Institute, Boston, MA, USA. ^[5] Division of Cardiology, University of California San Francisco, San Francisco, CA, USA. ^[6] Department of Epidemiology and Population Health, Stanford University School of Medicine, Stanford, CA, USA.;Stanford Cancer Institute, Stanford University School of Medicine, Stanford, CA, USA. ^[7]

DOI 10.1101/2024.04.15.24305836

PMID 38699369

发布时间 2024-10-23

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Improved multi-ancestry fine-mapping identifies <i>cis</i>-regulatory variants underlying molecular traits and disease risk.

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Improved multi-ancestry fine-mapping identifies <i>cis</i>-regulatory variants underlying molecular traits and disease risk.

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