Dual-specificity phosphatase 26 protects against kidney injury caused by ischaemia-reperfusion through restraint of TAK1-JNK/p38-mediated apoptosis and inflammation of renal tubular epithelial cells.
第一作者:
Heli,Xiang
第一单位:
Department of Kidney Transplant, Hospital of Nephrology, The First Affiliated Hospital of Xi'an Jiaotong University, Xi'an, 71006, China.
作者:
主题词
动物(Animals);再灌注损伤(Reperfusion Injury);细胞凋亡(Apoptosis);MAP激酶激酶激酶类(MAP Kinase Kinase Kinases);上皮细胞(Epithelial Cells);男(雄)性(Male);肾小管(Kidney Tubules);大鼠(Rats);p38丝裂原活化蛋白激酶类(p38 Mitogen-Activated Protein Kinases);大鼠, Sprague-Dawley(Rats, Sprague-Dawley);双特异性磷酸酶类(Dual-Specificity Phosphatases);细胞系(Cell Line);急性肾损伤(Acute Kidney Injury);炎症(Inflammation);MAP激酶信号系统(MAP Kinase Signaling System);丝裂原活化蛋白激酶磷酸酶类(Mitogen-Activated Protein Kinase Phosphatases);信号传导(Signal Transduction)
DOI
10.1016/j.taap.2024.116954
PMID
38705402
发布时间
2024-06-13
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