Phosphorylation of cell cycle and apoptosis regulatory protein-1 by stress activated protein kinase P38γ is a novel mechanism of apoptosis signaling by genotoxic chemotherapy.

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作者： Jaganathan,Venkatesh ^[1] ; Magesh,Muthu ^[1] ; Indulekha,Singaravelu ^[1] ; Vino T,Cheriyan ^[1] ; Sreeja C,Sekhar ^[1] ; Nuwan C P N,Acharige ^[2] ; Edi,Levi ^[3] ; Hadeel,Assad ^[4] ; Mary Kay H,Pflum ^[2] ; Arun K,Rishi ^[1]

作者单位： John D. Dingell V.A. Medical Center, Wayne State University, Detroit, MI, United States.;Karmanos Cancer Institute, Wayne State University, Detroit, MI, United States.;Department of Oncology, Wayne State University, Detroit, MI, United States. ^[1] Department of Chemistry, Wayne State University, Detroit, MI, United States. ^[2] John D. Dingell V.A. Medical Center, Wayne State University, Detroit, MI, United States.;Department of Pathology, Wayne State University, Detroit, MI, United States. ^[3] Karmanos Cancer Institute, Wayne State University, Detroit, MI, United States.;Department of Oncology, Wayne State University, Detroit, MI, United States. ^[4]

关键词 CCAR1/CARP-1 P38γ breast cancer genotoxic chemotherapy phosphorylation

DOI 10.3389/fonc.2024.1376666

PMID 38756656

发布时间 2024-09-18

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Frontiers in oncology

2024年14卷

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Phosphorylation of cell cycle and apoptosis regulatory protein-1 by stress activated protein kinase P38γ is a novel mechanism of apoptosis signaling by genotoxic chemotherapy.

Frontiers in oncology

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Frontiers in oncology

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Phosphorylation of cell cycle and apoptosis regulatory protein-1 by stress activated protein kinase P38γ is a novel mechanism of apoptosis signaling by genotoxic chemotherapy.

Frontiers in oncology

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Frontiers in oncology

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