Human-derived fecal microbiota transplantation alleviates social deficits of the BTBR mouse model of autism through a potential mechanism involving vitamin B<sub>6</sub> metabolism.

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第一作者： Lifeng,Zheng

第一单位： Brain Research Centre and Department of Neuroscience, Southern University of Science and Technology, Shenzhen, China.

作者： Lifeng,Zheng ^[1] ; Yinming,Jiao ^[2] ; Haolin,Zhong ^[3] ; Yan,Tan ^[1] ; Yiming,Yin ^[2] ; Yanhong,Liu ^[2] ; Ding,Liu ^[2] ; Manli,Wu ^[2] ; Guoyun,Wang ^[2] ; Jinqun,Huang ^[3] ; Ping,Wang ^[2] ; Meirong,Qin ^[4] ; Mingbang,Wang ^[4] ; Yang,Xiao ^[5] ; Tiying,Lv ^[6] ; Yangzi,Luo ^[7] ; Han,Hu ^[8] ; Sheng-Tao,Hou ^[9] ; Ling,Kui ^[2]

作者单位： Brain Research Centre and Department of Neuroscience, Southern University of Science and Technology, Shenzhen, China. ^[1] Xbiome Co. Ltd., Shenzhen, China. ^[2] Shenzhen Qianhai Shekou Free Trade Zone Hospital, Shenzhen, China. ^[3] Shenzhen Institute for Drug Control, Shenzhen, China. ^[4] Microbiome Therapy Center, South China Hospital, Medical School, Shenzhen University, Shenzhen, China. ^[5] Shanghai Key Laboratory of Birth Defects, Division of Neonatology, Children's Hospital of Fudan University, National Center for Children's Health, Shanghai, China. ^[6] Department of Hematology, Shenzhen Qianhai Shekou Free Trade Zone Hospital, Shenzhen, China. ^[7] Guangzhou University of Chinese Medicine, Guangzhou, China. ^[8] Faculty of Biology, Medicine and Health, The University of Manchester, Manchester, United Kingdom. ^[9]

关键词 BTBR Bacteroides autism spectrum disorder (ASD)fatty acid metabolism fecal microbiota transplantation (FMT)pyridoxal 5′-phosphate (PLP)social deficit vitamin B6 metabolism

主题词动物(Animals);小鼠(Mice);人类(Humans);疾病模型, 动物(Disease Models, Animal);维生素B6(Vitamin B 6);男(雄)性(Male);社会行为(Social Behavior);孤独性障碍(Autistic Disorder)

DOI 10.1128/msystems.00257-24

PMID 38780265

发布时间 2024-07-14

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2024年9卷6期

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Human-derived fecal microbiota transplantation alleviates social deficits of the BTBR mouse model of autism through a potential mechanism involving vitamin B<sub>6</sub> metabolism.

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Human-derived fecal microbiota transplantation alleviates social deficits of the BTBR mouse model of autism through a potential mechanism involving vitamin B<sub>6</sub> metabolism.

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