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Altered cortical synaptic lipid signaling leads to intermediate phenotypes of mental disorders.

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第一作者: Oliver,Tüscher
第一单位: Department of Psychiatry and Psychotherapy, University Medical Center of the Johannes Gutenberg-University Mainz, Mainz, Germany.;Leibniz Institute for Resilience Research Mainz, Mainz, Germany.;Institute for Molecular Biology Mainz, Mainz, Germany.
作者单位: Department of Psychiatry and Psychotherapy, University Medical Center of the Johannes Gutenberg-University Mainz, Mainz, Germany.;Leibniz Institute for Resilience Research Mainz, Mainz, Germany.;Institute for Molecular Biology Mainz, Mainz, Germany. [1] Department of Neurology, Johannes Gutenberg-University Mainz, Mainz, Germany.;Department of Neurology, Neural engineering with Signal Analytics and Artificial Intelligence (NESA-AI), University Hospital of Würzburg, Würzburg, Germany.;Informatics for Medical Technology, University Augsburg, Augsburg, Germany. [2] Department of Psychiatry and Psychotherapy, University Medical Center of the Johannes Gutenberg-University Mainz, Mainz, Germany. [3] Focus Program Translational Neuroscience, Johannes Gutenberg-University Mainz, Mainz, Germany.;Institute of Anatomy, University Medical Center Mainz, Mainz, Germany. [4] TARC, Translational Animal Research Center, University Medical Center of the Johannes Gutenberg University Mainz, Mainz, Germany. [5] Institute of Neurophysiology, University Medical Center, Goethe-University Frankfurt, Frankfurt, Germany. [6] Department of Molecular and Translational Neuroscience, Institute of Anatomy II, Cluster of Excellence-Cellular Stress Response in Aging-Associated Diseases (CECAD), Center of Molecular Medicine Cologne (CMMC), University of Cologne, Cologne, Germany. [7] Functional Imaging Unit, Diagnostic Radiology and Neuroradiology, University Medicine Greifswald, Greifswald, Germany. [8] Department of Psychiatry and Psychotherapy, University Medicine Greifswald, Greifswald, Germany. [9] Department SHIP/Clinical Epidemiological Research, Institute of Community Medicine, University Medicine Greifswald, Greifswald, Germany. [10] Institute for Translational Psychiatry, University of Münster, Münster, Germany.;Institute for Translational Neuroscience, University of Münster, Münster, Germany. [11] Institute for Translational Psychiatry, University of Münster, Münster, Germany. [12] Institute for Translational Neuroscience, University of Münster, Münster, Germany. [13] Institute of Pathophysiology, Johannes Gutenberg-University Mainz, Mainz, Germany. [14] Cognitive Neuroscience, Institute of Neuroscience and Medicine (IMN-3), Research Centre Jülich, Jülich, Germany. [15] Max Planck Institute of Metabolism Research, Cologne, Cluster of Excellence-Cellular Stress Response in Aging-Associated Diseases (CECAD), Cologne, Germany. [16] Department of Neurology, Johannes Gutenberg-University Mainz, Mainz, Germany. [17] Institute for Biomagnetism and Biosignalanalysis, University of Münster, Münster, Germany. [18] Institute for Translational Neuroscience, University of Münster, Münster, Germany. nitschr@uni-muenster.de. [19] Department of Neurology, Johannes Gutenberg-University Mainz, Mainz, Germany. johannes.vogt@uk-koeln.de.;Department of Molecular and Translational Neuroscience, Institute of Anatomy II, Cluster of Excellence-Cellular Stress Response in Aging-Associated Diseases (CECAD), Center of Molecular Medicine Cologne (CMMC), University of Cologne, Cologne, Germany. johannes.vogt@uk-koeln.de. [20]
DOI 10.1038/s41380-024-02598-2
PMID 38806692
发布时间 2024-11-09
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