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UCP1-dependent brown adipose activation accelerates cardiac metabolic remodeling and reduces initial hypertrophic and fibrotic responses to pathological stress.

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第一作者： Azariyas A,Challa

第一单位： Department of Internal Medicine, College of Medicine, Ohio State University, Columbus, Ohio, USA.

作者： Azariyas A,Challa ^[1] ; Pablo,Vidal ^[2] ; Santosh K,Maurya ^[3] ; Chandan K,Maurya ^[3] ; Lisa A,Baer ^[2] ; Yang,Wang ^[3] ; Natasha Maria,James ^[2] ; Parth J,Pardeshi ^[2] ; Matthew,Fasano ^[3] ; Andrew N,Carley ^[3] ; Kristin I,Stanford ^[2] ; E Douglas,Lewandowski ^[3]

作者单位： Department of Internal Medicine, College of Medicine, Ohio State University, Columbus, Ohio, USA. ^[1] Department of Internal Medicine, College of Medicine, Ohio State University, Columbus, Ohio, USA.;Davis Heart and Lung Research Institute, Ohio State University, Columbus, Ohio, USA.;Department of Physiology and Cell Biology, College of Medicine, Ohio State University, Columbus, Ohio, USA.;Department of Surgery, General and Gastrointestinal Surgery, College of Medicine, The Ohio State University, Columbus, Ohio, USA. ^[2] Department of Internal Medicine, College of Medicine, Ohio State University, Columbus, Ohio, USA.;Davis Heart and Lung Research Institute, Ohio State University, Columbus, Ohio, USA. ^[3]

关键词 UCP1 brown adipose tissue cardiac metabolic remodeling pathological hypertrophy pressure overload