CD52 knockdown inhibits aerobic glycolysis and malignant behavior of NSCLC cells through AKT signaling pathway.

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第一作者： Yini,Cai

第一单位： Department of Oncology, The Second Affiliated Hospital of Nanchang University; Jiangxi Key Laboratory of Clinical and Translational Cancer Research, 1 Minde Road, Nanchang, Jiangxi, China.

作者： Yini,Cai ^[1] ; Jiali,Zhao ^[1] ; Chen,Luo ^[1] ; Ming,Fang ^[2] ; Yanling,Yi ^[1] ; Yu,Chen ^[1] ; Peng,Huang ^[1] ; Lingmin,Liao ^[3] ; Long,Huang ^[1]

作者单位： Department of Oncology, The Second Affiliated Hospital of Nanchang University; Jiangxi Key Laboratory of Clinical and Translational Cancer Research, 1 Minde Road, Nanchang, Jiangxi, China. ^[1] Department of Yangxin People's Hospital of Hubei Province, 81 Ruxue Road, Xingguo Town, Yangxin County, Huangshi, Hubei, China. ^[2] Department of Ultrasound, The Second Affiliated Hospital, JiangXi Medical College, Nanchang University, Nanchang, China. ^[3]

关键词 AKT CD52 Migration NSCLC Proliferation aerobic glycolysis

DOI 10.7150/jca.86511

PMID 38817869

发布时间 2024-06-01

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