SHC-3: a previously unidentified C. elegans Shc family member functions in the insulin-like signaling pathway to enhance survival during L1 arrest.

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作者： Mercedes,Di Bernardo ^[1] ; Victoria L,León Guerrero ^[1] ; Jacob C,Sutoski ^[1] ; William Rod,Hardy ^[1] ; Lesley T,MacNeil ^[2]

作者单位： Department of Biochemistry and Biomedical Sciences, McMaster University, 1280 Main St W, Hamilton, ON L8S 4K1, Canada. ^[1] Department of Biochemistry and Biomedical Sciences, McMaster University, 1280 Main St W, Hamilton, ON L8S 4K1, Canada.;Farncombe Family Digestive Health Research Institute, McMaster University, 1280 Main St W, Hamilton, ON L8S 4K1, Canada.;Michael G. DeGroote Institute for Infectious Disease Research, McMaster University, 1280 Main St W, Hamilton, ON L8S 4K1, Canada. ^[2]

关键词 C. elegans DAF-16 DAF-2 L1 arrest SHC adapter protein development heat stress insulin-like signaling

主题词动物(Animals);秀丽新小杆线虫(Caenorhabditis elegans);漂亮新小杆线虫蛋白质类(Caenorhabditis elegans Proteins);叉头转录因子类(Forkhead Transcription Factors);胰岛素(Insulin);突变(Mutation);信号传导(Signal Transduction)

DOI 10.1093/genetics/iyae093

PMID 38861412

发布时间 2024-12-12

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