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Novel early-onset Alzheimer-associated genes influence risk through dysregulation of glutamate, immune activation, and intracell signaling pathways.

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第一作者： Carlos,Cruchaga

第一单位： Washington University, School of Medicine.

作者： Carlos,Cruchaga ^[1] ; Joseph,Bradley ^[2] ; Daniel,Western ^[3] ; Ciyang,Wang ^[4] ; Eder,Lucio Da Fonseca ^[5] ; Achal,Neupane ^[2] ; Jiji,Kurup ^[6] ; NIcholas,Ray ^[6] ; Melissa,Jean-Francois ^[5] ; Priyanka,Gorijala ^[4] ; Kristy,Bergmann ^[7] ; John,Budde ^[5] ; Eden,Martin ^[5] ; Margaret,Pericak-Vance ^[8] ; Michael,Cuccaro ^[9] ; Brian,Kunkle ^[10] ; John,Morris ^[4] ; David,Holtzman ^[2] ; Richard,Perrin ^[11] ; Adam,Naj ^[12] ; Jonathan,Haines ^[11] ; Gerard,Schellenberg ^[2] ; Victoria,Fernandez ^[6] ; Christiane,Reitz ^[5] ; Gary,Beecham ; Alzheimer’s Disease Genetics Consortium ; Charles F. and Joanne Knight Alzheimer’s disease research center Knight ADRC

作者单位： Washington University, School of Medicine. ^[1] Washington University in St. Louis. ^[2] Department of Psychiatry, Washington University School of Medicine, St. Louis, MO, USA. ^[3] Washington University School of Medicine. ^[4] University of Miami. ^[5] Columbia University. ^[6] Washington University School of Medicine, St. Louis. ^[7] The John P. Hussman Institute for Human Genomics, University of Miami, Miami, Florida. ^[8] University of Miami School of Medicine. ^[9] Knight Alzheimer Disease Research Center. ^[10] University of Pennsylvania. ^[11] Case Western Reserve University. ^[12]

DOI 10.21203/rs.3.rs-4480585/v1

PMID 38883718

发布时间 2026-05-19

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Novel early-onset Alzheimer-associated genes influence risk through dysregulation of glutamate, immune activation, and intracell signaling pathways.

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